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Amos Tanay

Pre-leukemic stem cells (top) with both mutated and healthy copies of the RUNX1 gene already display some of the characteristics of acute myeloid leukemia (AML). When the non-mutated copy of the gene is inactivated, disruptions in the spindle-assembly-checkpoint phase of cell division trigger cell death

Weizmann Institute researchers discover that a “standoff” between a mutated gene and its normal counterpart keeps certain cancer cells alive...

(l-r) Amir Bar, Dr. Amos Tanay, Netta Mendelson-Cohen, Prof. Varda Rotter, Dr. Zohar Mukamel, Naomi Goldfinger, Gilad Landan and Dr. Alina Molchadsky

Surprising changes in certain genomic markers may help explain how cells can turn cancerous as they age and divide

Dr. Amos Tanay

Dr. Amos Tanay was awarded the Krill Prize for Excellence in Scientific Research (2012)

(l-r) Gilad Landan, Dr. Amos Tanay and Rami Jaschek. New targets for treatment
Cancer-enhancing DNA may reside in a surprising part of the genome