Amos Tanay

Pre-leukemic stem cells (top) with both mutated and healthy copies of the RUNX1 gene already display some of the characteristics of acute myeloid leukemia (AML). When the non-mutated copy of the gene is inactivated, disruptions in the spindle-assembly-checkpoint phase of cell division trigger cell death

Leukemia Cells are Addicted to a Healthy Gene

29.09.2013

Cancer, Personalized Medicine, Molecular and Cell Biology, Genetics

Weizmann Institute researchers discover that a “standoff” between a mutated gene and its normal counterpart keeps certain cancer cells alive...

(l-r) Amir Bar, Dr. Amos Tanay, Netta Mendelson-Cohen, Prof. Varda Rotter, Dr. Zohar Mukamel, Naomi Goldfinger, Gilad Landan and Dr. Alina Molchadsky

How Good Cells Turn Bad

10.02.2013

Surprising changes in certain genomic markers may help explain how cells can turn cancerous as they age and divide

Dr. Amos Tanay

30.01.2012

Dr. Amos Tanay was awarded the Krill Prize for Excellence in Scientific Research (2012)

(l-r) Gilad Landan, Dr. Amos Tanay and Rami Jaschek. New targets for treatment

Found in the Desert

02.05.2010

Cancer, Genetics, Bioinformatics and Computational Biology

Cancer-enhancing DNA may reside in a surprising part of the genome

...

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This Far, No Further

Traces of Trauma in the Young Brain – and How to Erase Them

Scanning electron microscope images of the enamel layer enveloping mouse teeth

Newly Discovered Autoimmune Disorder Disrupts Tooth Enamel Development

Photo: Jonathan D. Muller

The Solar Forest

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This Far, No Further

Traces of Trauma in the Young Brain – and How to Erase Them

Newly Discovered Autoimmune Disorder Disrupts Tooth Enamel Development

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