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LATS1 knockout

What happens when genes in a cancer-preventing pathway are absent? 

The effects of p53 in cancer-associated fibroblasts on cancer cell migration

Reeducating cells in the tumor microenvironment reverses some of the malignancy

Krizhanovsky and group

A new method of counting cells could lead to future strategies for combating chronic inflammation

Prof. Moshe Oren

An Institute study finds a whole new role for one of the most famous proteins in cancer research

A partially unfolded protein (yellow) is broken down by a “scissor”-proteasome (blue and red)

Understanding how a pair of molecular “scissors” are kept in check may help treat disease

Genes that delay cell division exhibit reduced levels of expression (left) after exposure to a second, delayed pulse of growth factor. Those in cells receiving a single pulse (right box) maintained high expression levels

A gene that keeps cell division in check may hold clues to chemotherapy resistance. 

(l-r) Prof. Varda Rotter, Dr. Eldad Tzahor, Dr. Ariel Rinon, Alina Molchadsky and Dr. Rachel Sarig. Migration regulation

A gene that protects us from cancer is also involved in the timing of a crucial stage of embryonic development.

Eight Hours of Resistance

Why do cancer cells easily give in to the temptation to divide?

(l-r) Dr. Perry Stambolsky and Profs. Varda Rotter and Moshe Oren
Weizmann scientists celebrate three decades of research into the p53 tumor supressor research