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Chaperones Do the Two-Step

Understanding how certain proteins work as a team could shed new light on the way cells defend themselves against neurodegenerative disease

Argentinian postdocs

Working side by side on their postdocs, this couple feels right at home at Weizmann

(Bottom row, l-r) Prof. Ido Amit, Prof. Michal Schwartz and Dr. Hadas Keren-Shaul. (Top row, l-r) Dr. Assaf Weiner, Orit Matcovitch-Natan and Amit Spinrad

A newly discovered immune cell type may lead to a future treatment for Alzheimer's disease

Left: normal nerve cells; right cells genetically engineered to neutralize MTCH2. Fluorescent proteins reveal the calcium uptake in the mitochondria of these cells. The genetically engineered cells reveal dramatically less calcium uptake – evidence of the crucial role this gene plays in mitochondria function

Understanding how a gene studied in one lab affects the basic element researched in another may reveal what goes wrong in Alzheimer's 

 Inclusion bodies (red) are surrounded by lipid droplets (green)

Drops of fat could be key to keeping nerve cells healthy

Fluorescent image of Baker's yeast cells showing that the yeast Presenilin-like protein, Ypf1 (green), is found in the same cellular area as Presenilin, around the nucleus

Weizmann Institute researchers reveal an unusual cellular mechanism that may be faulty in Alzheimer’s disease

Dr. Yaniv Ziv

Dr. Yaniv Ziv want to know how our brain keeps memories of individual events in order

Microglia cells, obtained using a mouse model developed by Prof. Stephen Jung’s team

A new kind of genetic switch can target the activities of just one type of brain cell